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    Inactivation of Endothelial ADAM17 Reduces Retinal Ischemia-Reperfusion Induced Neurovascular Abnormalities

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    Authors
    Gutsaeva, Diana
    Lamiaa, Shalaby
    Wetzstein, Sara
    Thounaojam, Menaka
    Jadeja, Ravirajsinh
    Powell, Folami
    Martin, Pamela
    Kwok, Hang Fai
    Bartoli, Manuela
    URI
    http://hdl.handle.net/10675.2/623159
    
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    Abstract
    Retinal ischemia contributes to visual impairment in ischemic retinopathies such as diabetic retinopathy and other potentially blinding conditions. A member of the ADAM family of a disintegrin and metalloproteinases, ADAM17, contributes to multiple vascular pathologies through its ability to regulate inflammatory signals via ectodomain shedding. Here we investigated the specific contribution of endothelial ADAM17 to neurovascular damage associated with retinal ischemia using a mouse model of ischemiareperfusion (IR) injury.
    Affiliation
    Department of Ophthalmology
    Collections
    Department of Ophthalmology: Faculty Research and Presentations

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